[1]姜海平,皇甫姝辰.具核梭杆菌致病性与肠道屏障损伤研究进展[J].肠外与肠内营养杂志,2020,(02):65-68.[doi:10.16151/j.1007-810x.2020.02.001]
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具核梭杆菌致病性与肠道屏障损伤研究进展
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《肠外与肠内营养》杂志[ISSN:1007-810X/CN:32-1477/R]

卷:
期数:
2020年02期
页码:
65-68
栏目:
专家论坛
出版日期:
2020-03-10

文章信息/Info

作者:
姜海平皇甫姝辰
暨南大学附属第一医院胃肠外科,广东广州 510630
关键词:
具核梭杆菌 肠道屏障 肠道菌群
分类号:
R378
DOI:
10.16151/j.1007-810x.2020.02.001
文献标志码:
A

参考文献/References:


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[2] Guo L, Shokeen B, He X, et al. Streptococcus mutans SpaP binds to RadD of Fusobacterium nucleatum ssp. polymorphum. Mol Oral Microbiol, 2017, 32(5):355-364.
[3] Manson McGuire A, Cochrane K, Griggs AD, et al. Evolution ofinvasion in a diverse set of Fusobacterium species. mBio, 2014, 5(6):e01864.
[4] Strauss J, Kaplan GG, Beck PL, et al. Invasive potential of gut mucosa-derived Fusobacterium nucleatum positively correlateswith IBD status of the host. Inflamm Bowel Dis, 2011,17(9):1971-1978.
[5] Han YW , Ikegami A , Rajanna C , et al. Identification and Characterization of a Novel Adhesin Unique to Oral Fusobacteria.J Bacteriol, 2005, 187(15):5330-5340.
[6] Xu M , Yamada M , Li M , et al. FadA from Fusobacterium nucleatum Utilizes both Secreted and Nonsecreted Forms for Functional Oligomerization for Attachment and Invasion of Host Cells. J Biol Chem, 2007, 282(34):25000-25009.
[7] Han YW, Shi W, Huang GT, et al . Interactions between Periodontal Bacteria and Human Oral Epit helial Cells:Fusobacteriu m nucleat u m Adheres to and Invades Epit helial Cells. Infect Immu, 2000, 68(8):3140-3146.
[8] Brennan CA, Garrett WS. Gut Microbiota, Inflammation, and Colorectal Cancer. Annu Rev Microbiol, 2016, 70:395-411.
[9] Moore WE, Moore LV. The bacteria of periodontal diseases. Periodontol 2000, 1994, 5:66-77.
[10] Han YW, Shen T, Chung P, et al. Uncultivated bacteria as etiologic agents of intra-amniotic inflammation leading to preterm birth. J Clin Microbiol, 2009, 47(1):38-47.
[11] Wang X, Buhimschi CS, Temoin S, et al. Comparative microbial analysis of paired amniotic fluid and cord blood from pregnancies complicated by preterm birth and early-onset neonatal sepsis. PLoS One, 2013, 8(2):e56131.
[12] Tahara T, Shibata T, Kawamura T, et al. Fusobacterium Detected in Colonic Biopsy and Clinicopathological Features of Ulcerative Colitis in Japan. Dig Dis Sci, 2015, 60(1): 205-210.
[13] Sparks Stein P, Steffen MJ, Smith C, et al. Serum antibodies to periodontal pathogens are a risk factor for Alzheimer's disease. Alzheimers Dement, 2012, 8(3):196-203.
[14] Cao P, Chen Y, Chen Y, et al. Fusobacterium nucleatum Activates Endoplasmic Reticulum Stress to Promote Crohn's Disease Development via the Upregulation of CARD3 Expression. Front Pharmacol, 2020, 11:106.
[15] Fardini Y, Wang X, Témoin S, et al. Fusobacterium nucleatum adhesin FadA binds vascular endothelial cadherin and alters endothelial integrity. Mol Microbiol, 2011, 82(6):1468-1480.
[16] 王 昆, 贾银平, 朱 攀, 等 . 具核梭杆菌诱导的肠上皮细胞Caco-2炎症反应及凋亡. 免疫学杂志, 2015(4):313-317.
[17] Liu L, Liang L, Liang H, et al. Fusobacterium nucleatum Aggravates the Progression of Colitis by Regulating M1 Macrophage Polarization via AKT2 Pathway. Front Immunol,2019, 10:1324.
[18] Chen Y, Chen Y, Cao P, et al. Fusobacterium nucleatum facilitates ulcerative colitis through activating IL‐17F signaling to NF‐κB via the upregulation of CARD3 expression. J Pathol, 2020, 250(2):170-182.
[19] Bashir A, Miskeen AY, Hazari YM, et al. Fusobacterium nucleatum, inflammation, and immunity: the fire within human gut. Tumour Biol, 2016, 37(3): 2805-2810.
[20] Tavares LJ, de Avila ED, Klein MI, et al. Antimicrobial photodynamic therapy alone or in combination with antibiotic local administration against biofilms of Fusobacterium nucleatum and Porphyromonas gingivalis. J Photochem Photobiol B, 2018,188:135-145.
[21] Zhang T, Ying D, Qi M, et al. Anti-Biofilm Property of Bioactive Upconversion Nanocomposites Containing Chlorin e6 against Periodontal Pathogens. Molecules, 2019, 24(15):2692.

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备注/Memo

备注/Memo:
作者简介 :姜海平,主任医师,医学博士,从事胃肠外科及临床营养支持专业。E-mail:qwwer@139.com
更新日期/Last Update: 1900-01-01